The most frequent contributors to hereditary cancer risk in human population so far is the inherited mutations in the BRCA1 or BRCA2 tumor suppressor genes, often causing breast or ovarian cancer in young women of child-bearing age. Attempts to test the role that the BRCA genes play in regulating a repair process associated with genome duplication have proven frustratingly difficult in living mammalian cells. Now investigators at Beth Israel Deaconess Medical Center ...
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titled Mechanism of Cancer Caused by Loss of BRCA1 and BRCA2 Gene Function Identified.
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editor - Tuesday, April 29, 2014
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