The University of Texas MD Anderson Cancer Center scientists have identified the pancreatic cancer resistance mechanism. Pancreatic cancer addicted to mutant Kras signaling for their growth and progression have a ready-made substitute to tap if they're ever forced to go cold-turkey on the mutant oncogene. When researchers dialed up mutant Kras to spur pancreatic cancer growth in mice, and then shut it down, a group of recurrent tumors grew back independently ...
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Thursday, June 19, 2014
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