
A key mechanism that governs how cells of the epithelia shift between a rigid, highly structured and immobile state and a flexible and motile form has been identified by Ludwig Oxford researchers. Published in the current issue of iNature Cell Biology/i, their study shows that a tumor suppressor protein named ASPP2 functions as a molecular switch that controls this process and its reverse, both of which play a critical role in a number of biological phenomena, ...
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Monday, October 27, 2014
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