In a finding that could limit the damage done to the heart following a heart attack, researchers at UNC School of Medicine have found a way to convert fibroblasts, linked with the formation of scar tissue after an attack, into endothelial cells that can generate blood vessels to supply oxygen and nutrients to the injured regions of the heart. This switch is driven by p53, the well-documented tumor-suppressing protein. The UNC researchers showed that increasing the ...
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Thursday, October 16, 2014
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