Max Planck Institute scientists have discovered that mice deficient in Mfn2 gene die soon after birth. Therefore, the researchers suggest that lack of Mitofusin 2 affects mitochondrial function in other ways besides membrane fusion. The study findings may point to a new treatment for patients with diseases caused by loss of the mitochondrial protein. Mitofusin 2 and Mitofusin 1, located in the outer membrane of mitochondria, are required for mitochondrial fusion, ...
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titled Coenzyme Q Supplements Might Help Treat Patients With Diseases Caused by Mfn2 Mutations.
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Monday, February 16, 2015
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